Chronic diabetic wounds, including diabetic foot ulcers, are a significant burden for patients, as impaired blood vessel growth hinders the healing process. A recent breakthrough offers hope by combining small extracellular vesicles (sEVs) loaded with miR-221-3p and a GelMA hydrogel to target thrombospondin-1 (TSP-1), a protein that suppresses angiogenesis. This new bioactive wound dressing not only accelerates healing but also promotes blood vessel formation, offering a promising new…
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News Source: www.sciencedaily.com
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